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Faculty & Staff

Areas of Expertise

Neurobiology

Degrees

PhD, Neuroscience, University of Virginia, 1991

Professional Publications & Contributions

• Pollack, A.E., Exploring the complexities of experimental design: Using an on-line reaction time program as a teaching tool for diverse student populations. Journal of Undergraduate Neuroscience Education (JUNE), Fall 2010, 9(1): A47-A50.
• Pollack, A.E., *Dimitrov, K.D., and *Drake, J.D. Prior treatment (priming) with caffeine sensitizes D2-dopamine mediated contralateral rotational behavior in 6-hydroxydopamine lesioned rats. Pharmacology 86: (2010) 73-78.
• Pollack, A.E. and *Thomas, L.T. D1 priming enhances both D1- and D2-mediated rotational behavior and striatal Fos expression in 6-hydroxydopamine lesioned rats. Pharmacol Biochem Behav. 94: (2010) 346-351.
• Pollack, A.E., and *Angerer, M.R. Muscarinic receptor blockade attenuates reserpine-mediated Fos induction in the rat striatopallidal pathway. Brain Res. 1058: (2005) 189-192.
• Pollack, A.E., *St. Martin, J.L., and *MacPherson, A.T. Role of NMDA and AMPA glutamate receptors in the induction and the expression of dopamine-mediated sensitization in 6-hydroxydopamine lesioned rats. Synapse 56: (2005) 45-53.
• Pollack, A.E., and *Haisley, E.C. NMDA glutamate receptor stimulation is required for the expression of D2 dopamine mediated responses in apomorphine primed 6-hydroxydopamine lesioned rats. Brain Res. 897: (2001) 213-216.
• Pollack, A.E., and *Strauss, J.B. Time dependence and role of NMDA glutamate receptors in the priming of D2-mediated rotational behavior and striatal Fos expression in 6-hydroxydopamine lesioned rats. Brain Res. 827: (1999) 160-168.
• Pollack, A.E., and *Yates, T.M. Prior D1 dopamine receptor stimulation is required to prime D2-mediated striatal Fos expression in 6-hydroxydopamine lesioned rats. Neuroscience 94: (1999) 505-514.
• Pollack, A.E., *Bird, J.L., *Lambert, E.B., *Florin, Z.P., and *Castellar, V.L. Role of NMDA glutamate receptors in regulating D2 dopamine-dependent Fos induction in the rat striatopallidal pathway. Brain Res. 818: (1999) 543-547.
• Pollack, A.E., Turgeon, S.M., and Fink, J.S. Apomorphine-priming alters the response of striatal outflow pathways to D2 agonist stimulation in 6-hydroxydopamine lesioned rats. Neuroscience 79: (1997) 70-93.
• Pollack, A.E., and Fink, J.S. Synergistic interaction between an adenosine antagonist and a D1 dopamine agonist on rotational behavior and striatal c-Fos induction in 6-hydroxydopamine lesioned rats. Brain Res. 743: (1996) 124-130.
• Pollack, A.E., and Fink, J.S. Adenosine antagonists potentiate D2 dopamine-dependent activation of Fos in the striatopallidal pathway. Neuroscience 68: (1995) 721-728.
• Pollack, A.E. Exploring the complexities of experimental design: using an on-line reaction time program as a teaching tool for diverse student populations. Journal for Undergraduate Neuroscience Education (JUNE) 9:(2010) A47-A50.
• Pollack, A.E. Media Review, The Neuron Connection: Modeling Parkinson’s Disease. Journal for Undergraduate Neuroscience Education (JUNE) 5: (2006) R7-R8.
• Pollack, A. Co-activation of D1 and D2 dopamine receptors: in marriage, a case of his, hers and theirs. Science STKE, 255: (2004) pe50.
• Brown, C., and Pollack, A. Reconstructing the paradigm: Teaching across the disciplines. Journal of Undergraduate Neuroscience Education (JUNE) 3: (2004) A9-A15.
• Pollack, A.E. Basal Ganglia: Anatomy, Physiology and Pharmacology. Neurologic Clinics 19: (2001) 523-534.

Additional Information

Professional Appointments
Associate Professor, Department of Biology, University of Massachusetts-Boston, 2006-
Assistant Professor, Department of Biology, University of Massachusetts-Boston, 2000-2006
Assistant Professor, Department of Psychology, Brown University, 1995-2000

Teaching
Seminar in Neurobiology (Bio 693)
Neurobiology (Bio316/318)
Biology of Life (Bio101)
Addiction (Honors Program)